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Re: [IP] Re: Large meals and delayed digestion



You might want to check out this very interesting page:

http://www.bio.ilstu.edu/rubinlab/endocrinology/lectures/Lecture_
09-Metabolism,_GI.pdf

e. CCKis secreted by the Endocrine cells of the small intestine
in response tocertain amino acids (Tryptophan and Phenylalanine),
lipids andfatty acidsi. CCK contracts the gall bladder &
stimulates the release ofpancreatic enzymes, and stimulates the
release of glucagonii. CCK enhances the action of secretin in
stimulating bicarbonate releasefrom the pancreas and it delays
gastric emptyingiii. CCK is one of the hormones which initiates
Satiety(full feelingduring eating). InsulinandLeptinincrease the
sensation offeeling fulle. CCKis secreted by the Endocrine cells
of the small intestine in response tocertain amino acids
(Tryptophan and Phenylalanine), lipids andfatty acidsi. CCK
contracts the gall bladder & stimulates the release ofpancreatic
enzymes, and stimulates the release of glucagonii. CCK enhances
the action of secretin in stimulating bicarbonate releasefrom the
pancreas and it delays gastric emptyingiii. CCK is one of the
hormones which initiates Satiety(full feelingduring eating).
InsulinandLeptinincrease the sensation offeeling fulle. CCKis
secreted by the Endocrine cells of the small intestine in
response tocertain amino acids (Tryptophan and Phenylalanine),
lipids andfatty acidsi. CCK contracts the gall bladder &
stimulates the release ofpancreatic enzymes, and stimulates the
release of glucagonii. CCK enhances the action of secretin in
stimulating bicarbonate releasefrom the pancreas and it delays
gastric emptyingiii. CCK is one of the hormones which initiates
Satiety(full feelingduring eating). InsulinandLeptinincrease the
sensation offeeling fullScroll down to '5 e' (pay particularly
close attention to points i and iii.) on page 2 and read about
CCK (cholecystokinin) - sounds like the hormone Dr Bernstein was
describing! This would probably explain what Ryan experienced.

Oh what the heck...here it is:
e. CCK is secreted by the Endocrine cells of the small intestine
in response to certain amino acids (Tryptophan and
Phenylalanine), lipids and fatty acids
i. CCK contracts the gall bladder & stimulates the release of
pancreatic enzymes, and stimulates the release of glucagon
ii. CCK enhances the action of secretin in stimulating
bicarbonate release from the pancreas and it delays gastric
emptying
iii. CCK is one of the hormones which initiates Satiety (full
feeling during eating). Insulin and Leptin increase the sensation
of feeling full.

Take care, Kerri, mom to Shannon-12.5, dx 11/96, pumping 11/99,
and 7 other blessings :)

Wayne said:
Shawna, I've read  this explanation of Bernstein's before, and I
agree that it sounds reasonable.  Unfortunately he's just made up
a lot of the physiology to suit his imagination.  He imagines
mythical horomones that cause the pancreas to release glucagon.
In addition to there being this undefined mythical hormone,
glucagon release tends to work pretty quickly, but we know this
"stuffed" effect on BG often lasts many hours.  Type 1s also have
been reported to have impaired glucaogon responses.  Now maybe if
Bernstein actually did an experiment eating sawdust and measuring
his BG over the next few hours, I'd believe him more :-)
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